Abstract ↔ claims

run not observed · v1 provisional awaiting approval

Which claims does the abstract carry, and which does it drop?

for Computational modelling identifies key determinants of subregion-specific dopamine dynamics in the striatum · this layer across all papers · json

Provisional

This layer needs a corpus-scope decision that has not been ruled on yet, so what it produces would change if the decision changed. It waits on claim-format, relation-vocab.

Awaiting approval

Waiting for approval. That is a statement about the record, not about whether anyone has read this: people read the corpus without stamping what they read, and only a stamp leaves a trace. Approval is an operation on a version, not a step of its own — it is recorded against the version it was granted to, so running this layer again does not carry it forward.

What this layer produced

Abstract mapped to claims

The paper's abstract is shown with each sentence linked to the claim(s) it represents in the dependency graph. Hover or click a sentence to highlight the corresponding claim cards. Below: what the graph contains that the abstract leaves out, and vice versa.

Abstract

1Striatal dopamine (DA) release regulates reward-related learning and motivation and is believed to consist of a short-lived phasic and continuous tonic component. 2Here, we build a large-scale three-dimensional model of extracellular DA dynamics in dorsal (DS) and ventral striatum (VS). 3The model predicts rapid dynamics in DS with little to no basal DA and slower dynamics in the VS enabling build-up of tonic DA levels. 4These regional differences do not reflect release-related phenomena but rather differential dopamine transporter (DAT) activity. 5Interestingly, our simulations posit DAT nanoclustering as a possible regulator of this activity. 6Receptor binding simulations show that D1 receptor occupancy follows extracellular DA concentration with milliseconds delay, while D2 receptors do not respond to brief pauses in firing but rather integrate DA signal over seconds. 7Summarised, our model distills recent experimental observations into a computational framework that challenges prevailing paradigms of striatal DA signalling.

[1]
background / framing — not a paper-specific claim
[2]
background / framing — not a paper-specific claim
[3]
combines multiple claims → H3.1 · During 4 Hz pacemaker activity, the dorsal striatum produces partially segregate, H3.6 · With DAT Vmax reduced to 33% of DS and terminal density at 90%, VS produces a di
[4]
synthesis across claims → H3 · Regional differences in striatal DA dynamics (DS hotspots vs VS pervasive tonic , H3.4 · Across parameter sweeps of active terminal fraction, quantal size, release proba, C1 · VMAT2 immunostaining shows no significant dorsoventral gradient in striatum (p=0
[5]
synthesis across claims → H2 · DAT nanoclustering is a possible regulator of effective transporter activity, wi, H2.2 · Dense DAT nanoclusters (20 nm diameter) take approximately 400 ms to clear a 100, H2.1 · Super-resolution dSTORM imaging shows DAT is significantly more nanoclustered in
[6]
combines multiple claims → H1 · D1 and D2 receptors operate on distinct temporal scales — D1 tracks burst DA wit, H1.1 · D1R occupancy closely tracks extracellular DA with approximately 50 ms delay dur, H1.3 · D2R occupancy takes at least 5 s to return to baseline after a burst due to slow, H1.2 · A complete 1 s pause in firing reduces D2R occupancy from approximately 0.55 to
[7]
background / framing — not a paper-specific claim
Claims in the graph not surfaced in the abstract
  • ds-vs-vmax-ratio-assumed fig2A (implied throughout)
    The 3:1 DS:VS DAT Vmax ratio (DS = 6 µM·s⁻¹, VS = 2 µM·s⁻¹) is assumed from published literature rather than directly measured in this study; the immunostaining gradient (Figure 2—supplement 1) corroborates this assumption at the protein level but does not directly establish the functional Vmax ratio.
  • M1 d2r-initialization-unjustified fig1H
    D2 receptor occupancy is initialized at 0.4 in all receptor dynamics simulations without derivation from steady state; at the modeled EC50 of 7 nM and simulated tonic [DA] of ~10 nM in DS, equilibrium occupancy would be approximately 0.59. No sensitivity analysis over this initialization is reported.
  • Sc2 nanoclustering-model-varicosity-scale fig4C, fig4D, fig4E, fig4F
    The nanoclustering simulations (Figure 4C–F) operate in a standalone varicosity-scale model (1.8 × 1.8 µm domain, 0.02 µm voxels) architecturally separate from the tissue-level model used in Figures 1–3 (100 µm domain, 1 µm voxels); no formal coupling exists between the two models and no effective-Vmax output from the nanoclustering simulation feeds into the tissue simulation.
  • Sc1 nanoclustering-constant-vmax-constraint fig4C, fig4D, fig4E, fig4F
    The nanoclustering simulations hold total DAT Vmax constant across clustered and unclustered conditions — the per-voxel rate is multiplied by a normalization factor so total integrated uptake capacity is identical; if DAT nanoclustering co-occurs with increased total DAT expression in biology, the clearance-slowing result would not hold.
  • E3 low-burst-no-spillover-high-burst-does fig1G
    3 APs at 10 Hz generates no significant DA spillover outside the burst zone; 6 APs at 20 Hz and 12 APs at 40 Hz cause frequency-dependent spillover exposing 10× and 30× the burst volume to concentrations above 100 nM respectively.
  • E1 d2r-occupancy-higher-in-vs fig2G
    D2R occupancy during pacemaker activity is approximately 0.8 in VS versus approximately 0.55 in DS, consistent with higher prevailing tonic DA in VS.
  • E4 vs-lowest-percentiles-above-10nm fig2D
    Even the lowest DA concentration percentiles in VS exceed 10 nM during 4 Hz pacemaker activity.
  • H3.2 fscv-matches-may-wightman-1989 fig2E
    Simulated FSCV responses to 10, 30, and 60 Hz stimulation closely replicate May & Wightman (1989): VS reaches considerably higher peak DA than DS at all three stimulation frequencies.
  • H3.3 vmax-modulation-larger-impact-in-vs fig3K
    A ±50% change in DAT Vmax shifts tonic DA by 38 nM in VS but only 11 nM in DS, indicating VS operates closer to the Km saturation regime and is more sensitive to DAT modulation.
  • H3.5 vs-low-active-fraction-resembles-ds-distribution fig3B
    VS at 5% active terminals produces a spatial DA distribution resembling DS at 100% active terminals, demonstrating VS operates in a low-focality high-coverage regime while DS requires dense terminal participation for equivalent spatial reach.
  • E2 dat-immunostaining-dorsoventral-gradient fig2—supplement 1B, fig2—supplement 1C
    DAT expression is significantly higher in dorsal than ventral striatum (p=0.0021, one-sided t-test, n=4 mice), corroborating the 3:1 Vmax ratio assumed in the model.

How it is defined

A model answers this layer, so the prompt is the layer. It is reproduced below from the committed file, and it is a declared input — editing it makes every run that used it stale.

extract/prompts/abstract-map.mdthe prompt it runs undernot in the repository

The declaration names this path and the repository does not have it. An input that does not exist hashes to nothing, so it cannot make a run stale — the layer is declared to depend on something it is not in fact tracking.

Artifacts

Versions

From the run ledger. There is no changelog beside it to keep in step.

  1. v1 · 2026-04-20 · unrecorded backfilled from the artifact

    backfilled from the artifact on disk

This layer across the corpus

Across the corpus

9 run not observed · 1 stale·a paper links to its own cell, where this layer's output for it is rendered

PaperStateVersionLast runOutputCell
A three-dimensional immunofluorescence atlas of the …backfilled from the artifact on diskrun not observedv12026-04-20artiushin-2026-spider-atlas.jsonjson
Distinct representational properties of cues and con…backfilled from the artifact on diskrun not observedv12026-04-20bouyeure-2026-fear-rsa.jsonjson
Computational modelling identifies key determinants …backfilled from the artifact on diskrun not observedv12026-04-20ejdrup-2026-dopamine.jsonjson
Contributions of insula and superior temporal sulcus…re-run for the current treestalev52026-09-12gadeke-2026-guilt-insula.jsonjson
Spatially targeted inhibitory rhythms differentially…backfilled from the artifact on diskrun not observedv12026-04-19headley-2026-inhibitory-rhythms.jsonjson
Feedback of peripheral saccade targets to early fove…backfilled from the artifact on diskrun not observedv12026-04-19kammer-2026-foveal-feedback.jsonjson
iGABASnFR2 is an improved genetically encoded protei…backfilled from the artifact on diskrun not observedv12026-04-20kolb-2026-igabasnfr2.jsonjson
A deep learning pipeline for mapping in situ network…backfilled from the artifact on diskrun not observedv12026-04-20rozak-2026-neurovascular-dl.jsonjson
Self-association enhances early attentional selectio…backfilled from the artifact on diskrun not observedv12026-04-20scheller-2026-self-prioritization.jsonjson
Impaired excitability of fast-spiking neurons in a n…backfilled from the artifact on diskrun not observedv12026-04-20wengert-2026-kcnc1.jsonjson

Inputs and outputs

Reads, besides its dependencies
Produces
  • site/src/data/abstract-mapping/{paper}.json

One per paper — the table above links each one that exists.

Views
  • document — rendered above, from the artifact itself
  • comparison — on the cell page, two versions aligned by the matcher, wherever the ledger holds more than one

Running it

The command comes from the declaration, so this text and what actually runs cannot diverge. pipeline.py run also runs the unmet dependencies first.

python3 scripts/pipeline.py run <paper> abstract-map

Underneath, that runs cd extract && python3 -m claim_graphs.cli abstract-map --paper {paper}.