Abstract ↔ claims

run not observed · v1 provisional awaiting approval

Which claims does the abstract carry, and which does it drop?

for A deep learning pipeline for mapping in situ network-level neurovascular coupling in multi-photon fluorescence microscopy · this layer across all papers · json

Provisional

This layer needs a corpus-scope decision that has not been ruled on yet, so what it produces would change if the decision changed. It waits on claim-format, relation-vocab.

Awaiting approval

Waiting for approval. That is a statement about the record, not about whether anyone has read this: people read the corpus without stamping what they read, and only a stamp leaves a trace. Approval is an operation on a version, not a step of its own — it is recorded against the version it was granted to, so running this layer again does not carry it forward.

What this layer produced

Abstract mapped to claims

The paper's abstract is shown with each sentence linked to the claim(s) it represents in the dependency graph. Hover or click a sentence to highlight the corresponding claim cards. Below: what the graph contains that the abstract leaves out, and vice versa.

Abstract

1Functional hyperemia is a well-established hallmark of healthy brain function, whereby local brain blood flow adjusts in response to a change in the activity of the surrounding neurons. 2Although functional hyperemia has been extensively studied at the level of both tissue and individual vessels, vascular network-level coordination remains largely unknown. 3To bridge this gap, we developed a deep learning-based pipeline that uses two-photon fluorescence microscopy images of cerebral microcirculation to enable automated reconstruction and quantification of the geometric changes across the microvascular network, comprising hundreds of interconnected blood vessels, pre and post-activation of the neighboring neurons. 4The pipeline’s utility was demonstrated in the Thy1-ChR2 optogenetic mouse model, where we observed network-wide vessel radius changes to depend on the photostimulation intensity, with both dilations and constrictions occurring across the cortical depth, at an average of 16.1±14.3 μm (mean ± SD) away from the most proximal neuron for dilations; and at 21.9±14.6 μm away for constrictions. 5We observed a significant heterogeneity of the vascular radius changes within vessels, with radius adjustment varying by an average of 24 ± 28% of the resting diameter, likely reflecting the heterogeneity of the distribution of contractile cells on the vessel walls. 6A graph theory-based network analysis revealed that the assortativity of adjacent blood vessel responses rose by 152 ± 65% at 4.3 mW/mm² of blue photostimulation vs. the control, with a 4% median increase in the efficiency of the capillary networks during this level of blue photostimulation in relation to the baseline. 7Interrogating individual vessels is thus not sufficient to predict how the blood flow is modulated in the network. 8Our pipeline, enables tracking of the microvascular network geometry over time, relating caliber adjustments to vessel wall-associated cells’ state, and mapping network-level flow distribution impairments in experimental models of disease.

[1]
no corresponding claim in the graph
[2]
no corresponding claim in the graph
[3]
no corresponding claim in the graph
[4]
direct map → H1.P2.8 · Vessel radius adjustments during optogenetic stimulation show 24 ± 28% variation, H1.P2.6 · Following ChR2 optogenetic activation at 458 nm, capillary dilations occur on av, H1.P2.5 · Constricting capillaries are located on average 37±179 µm deeper in cortex than , H1.P2 · Under ChR2 photostimulation we should see a dilation-vs-constriction spatial gra
[5]
direct map → H1.P2.2 · Capillary radius varies along vessel length by 24±28% of the mean resting radius, H1.P2.8 · Vessel radius adjustments during optogenetic stimulation show 24 ± 28% variation, S1 · Single-vessel measurements cannot predict network-level blood flow modulation: w
[6]
direct map → H1.P2.7 · Vascular network assortativity increases by 152 ± 65% at 4.3 mW/mm² optogenetic , H1.P2.4 · Capillary network efficiency shows a median 4% increase during peak optogenetic , H1.P2.3 · Capillary dilations following 458 nm ChR2 stimulation (0.90±0.93 µm at 1.1 mW/mm
[7]
synthesis across claims → S1 · Single-vessel measurements cannot predict network-level blood flow modulation: w, H2 · Optogenetic neuronal activation drives coordinated, network-level vascular respo
[8]
synthesis across claims → H1 · A DL segmentation + registration + graph-analysis pipeline can deliver automated, Sc4 · Pipeline-and-application scope: one DL stack trained on Thy1-ChR2-EYFP cranial-w
Claims in the graph not surfaced in the abstract
  • H1.P1.4 unetr-outperforms-ilastik-hd95 fig3
    UNETR ensemble segmentation shows significantly better HD95 surface distance than ilastik for both vessel and neuron channels (p<0.05 Wilcoxon signed-rank), while ilastik over-segments vessels with high recall (0.89±0.19) but low precision (0.37±0.33), evaluated on nine test images (507×507×250 µm) from six held-out mice.
  • H1.P2.1 artery-dilates-venule-unchanged-at-low-power fig7A
    At 1.1 mW/mm² 458 nm stimulation, a sample artery dilated 1.33±0.86 µm (p<1e-4) and a sample capillary dilated 0.42±0.39 µm (p<1e-4), while a sample venule showed no significant radius change (p=0.22), demonstrating vessel-type heterogeneity in optogenetic neurovascular responses.
  • H1.P2.9 wt-controls-no-blue-green-difference app1fig9
    Wild-type C57BL/6J mice (n=4) show no statistically distinguishable capillary radius distributions following blue (458 nm) versus green (552 nm) photostimulation, confirming that vascular responses in Thy1-ChR2 mice are ChR2-specific and not attributable to photothermal or non-specific light effects.
  • E1 novas3d-generalizes-qualitatively-ood app1fig12, app1fig13
    The NOVAS3D segmentation model produces qualitatively reasonable vessel segmentations on out-of-distribution data including a different mouse strain (C57BL/6), a different species (Fischer rat), and a different microscope modality (light-sheet fluorescence microscopy, Miltenyi UltraMicroscope Blaze), without retraining.
  • Sc3 responder-threshold-2sd-untested app1fig14
    Vessels are classified as responders if their radius change exceeds twice the baseline standard deviation (2×σ); this threshold is not sensitivity-tested in the main analysis, though an alternative 10% threshold is shown in Appendix 1—figure 14 with qualitatively similar results.

How it is defined

A model answers this layer, so the prompt is the layer. It is reproduced below from the committed file, and it is a declared input — editing it makes every run that used it stale.

extract/prompts/abstract-map.mdthe prompt it runs undernot in the repository

The declaration names this path and the repository does not have it. An input that does not exist hashes to nothing, so it cannot make a run stale — the layer is declared to depend on something it is not in fact tracking.

Artifacts

Versions

From the run ledger. There is no changelog beside it to keep in step.

  1. v1 · 2026-04-20 · unrecorded backfilled from the artifact

    backfilled from the artifact on disk

This layer across the corpus

Across the corpus

9 run not observed · 1 stale·a paper links to its own cell, where this layer's output for it is rendered

PaperStateVersionLast runOutputCell
A three-dimensional immunofluorescence atlas of the …backfilled from the artifact on diskrun not observedv12026-04-20artiushin-2026-spider-atlas.jsonjson
Distinct representational properties of cues and con…backfilled from the artifact on diskrun not observedv12026-04-20bouyeure-2026-fear-rsa.jsonjson
Computational modelling identifies key determinants …backfilled from the artifact on diskrun not observedv12026-04-20ejdrup-2026-dopamine.jsonjson
Contributions of insula and superior temporal sulcus…re-run for the current treestalev52026-09-12gadeke-2026-guilt-insula.jsonjson
Spatially targeted inhibitory rhythms differentially…backfilled from the artifact on diskrun not observedv12026-04-19headley-2026-inhibitory-rhythms.jsonjson
Feedback of peripheral saccade targets to early fove…backfilled from the artifact on diskrun not observedv12026-04-19kammer-2026-foveal-feedback.jsonjson
iGABASnFR2 is an improved genetically encoded protei…backfilled from the artifact on diskrun not observedv12026-04-20kolb-2026-igabasnfr2.jsonjson
A deep learning pipeline for mapping in situ network…backfilled from the artifact on diskrun not observedv12026-04-20rozak-2026-neurovascular-dl.jsonjson
Self-association enhances early attentional selectio…backfilled from the artifact on diskrun not observedv12026-04-20scheller-2026-self-prioritization.jsonjson
Impaired excitability of fast-spiking neurons in a n…backfilled from the artifact on diskrun not observedv12026-04-20wengert-2026-kcnc1.jsonjson

Inputs and outputs

Reads, besides its dependencies
Produces
  • site/src/data/abstract-mapping/{paper}.json

One per paper — the table above links each one that exists.

Views
  • document — rendered above, from the artifact itself
  • comparison — on the cell page, two versions aligned by the matcher, wherever the ledger holds more than one

Running it

The command comes from the declaration, so this text and what actually runs cannot diverge. pipeline.py run also runs the unmet dependencies first.

python3 scripts/pipeline.py run <paper> abstract-map

Underneath, that runs cd extract && python3 -m claim_graphs.cli abstract-map --paper {paper}.